色吊丝永久性观看网站-中出内射颜射骚妇-激情偷乱人成视频在线观看-亚洲熟女乱色综合亚洲小说

Established at diagnosis of a 68-year-old male with acute myeloid leukemia (AML M6) secondary to myelodysplastic syndrome (MDS, subtype refractory anemia with excess of blasts, RAEB) in 1989; Cells were described to be proliferatively responsive to GM-CSF and IL-3. Cells have been shown to synthesize haemoglobin when GM-CSF or IL-3 is substituted by erythropoietin (Epo). They have been reported to be positive for leukocyte common antigen (CD45) and some multilineage markers such as CD13, CD33 and CD34, but are negative for T- and B-cell antigens and mature myelomonocytic antigens. Due to their reaction with some monoclonal antibodies recognising erythroid and platelet glycoproteins it has been suggested that F-36P has a multilineage phenotype.

于1989年診斷1例68歲男性急性髓系白血病(AML M6)繼發(fā)于骨髓增生異常綜合征(MDS，亞型難治性貧血，母細胞過多，RAEB);細胞被描述為對GM-CSF和IL-3有增殖反應。細胞合成血紅蛋白時GM-CSF或IL-3被紅細胞生成素(Epo)取代。據(jù)報道，它們對白細胞共同抗原(CD45)和一些多系標志物如CD13、CD33和CD34呈陽性反應，但對T、b細胞抗原和成熟的髓單核細胞抗原呈陰性反應。由于其與一些識別紅細胞和血小板糖蛋白的單克隆抗體的反應，已被認為F-36P具有多系表型。